By Sunil R. Lakhani, Caroline J. Finlayson, Susan A. Dilly, Mitesh Gandhi
This re-creation is an easy advent to pathology and the mechanisms of affliction. It places pathology into its ancient, clinical and medical context for all clinical, dental and nursing scholars and different future health professionals.
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Extra resources for Basic pathology : an introduction to the mechanisms of disease
Coli LT1 and LT2. A:B types are encountered in diphtheria, botulism and tetanus. tetani and C. botulinum, which cause tetanus and botulism respectively. These diseases are purely due to toxin-mediated action following infection and are quite different in pathology, yet the molecular action of the two toxins is identical. They are both endopeptidases specific for synaptobrevin, a protein found in the cytoplasm of synaptic vesicles. However, the binding (B) domains of the toxins show different specificities for cell receptors.
It causes fever and macrophage 33 34 Chapter 2 What causes disease? 2 Simple summary of the ways of classifying bacteria Gram-positive Examples of aerobic and anaerobic bacteria Gram-negative Acid fast Obligate aerobes Bacillus cereus Neisseria Mycobacteria Facultative anaerobes Bacillus anthracis Staphylococcus E. coli Salmonella Microaerophilic bacteria Streptococcus Spirochaetes Obligate anaerobes Clostridia Bacteroides Examples of morphologically distinct Gram-positive and Gram-negative bacteria Cocci (spherical) Streptococci (in chains) Staphylococci (in clusters) Neisseria Bacilli (rod-shaped) Corynebacteria Clostridia Bacillus Haemophilus Bordetella Klebsiella, Proteus, Shigella, E.
G. g. E. g. phospholipase C activity toxin Clostridium perfringens, spore-forming toxins (thiol-activating haemolysins) such as streptolysin (S. pyogenes), pnemolysin (S. These can be grouped by the type of enzymic activity: – ADP-ribosylation (cholera, diphtheria, pertussis) 35 36 Chapter 2 What causes disease? – N-glycosidases (shiga toxin) – glucosyl transferases (C. difficile toxin A and B) – Zn2ϩ-requiring endopeptidases (tetanus and botulism toxins) GTP binding proteins are often the target for ADPribosylation by type III bacterial exotoxins.